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BAM-15

Moderate Research

Mitochondrial Uncoupler | Metabolic Enhancer

Dose 25-50 mg oral
Frequency Once or twice daily with meals
Cycle 4-8 weeks based on research protocols
Storage Room temperature, protect from light and moisture

BAM-15 is a synthetic mitochondrial uncoupler that has emerged as a promising research compound for obesity and metabolic disorders. Unlike traditional uncouplers like DNP which have serious toxicity concerns, BAM-15 demonstrates a superior safety profile while effectively increasing energy expenditure and fat oxidation. Research in mice shows BAM-15 reduces body fat without affecting food intake, lean mass, or body temperature. It is approximately 7-fold more potent than DNP and does not induce the dangerous hyperthermia associated with older uncouplers. Note: BAM-15 is a small molecule compound, not a peptide, but is commonly sold alongside peptide products.

Mechanism of Action

BAM-15 targets the inner mitochondrial membrane, enhancing proton permeability and dissipating the proton gradient. This uncouples electron transport from ATP synthesis, forcing mitochondria to increase respiration and burn more substrates (particularly fat) to maintain energy production. BAM-15 activates AMP-activated protein kinase (AMPK) in response to ATP depletion, promoting glucose uptake and fatty acid oxidation. It also activates PGC-1α (peroxisome proliferator-activated receptor gamma coactivator 1-alpha), enhancing mitochondrial biogenesis. Unlike DNP or FCCP, BAM-15 does not depolarize plasma membranes or induce apoptosis at effective concentrations, explaining its improved safety profile.

Key Benefits

  • Increases fat oxidation without affecting food intake
  • Reduces body fat mass in research models
  • 7-fold more potent than DNP with better safety
  • Does not cause dangerous hyperthermia
  • No effect on lean body mass
  • Improves insulin sensitivity
  • Orally bioavailable
  • Activates AMPK and PGC-1α pathways
  • Synergistic with GLP-1 agonists (semaglutide)
Molecular Weight
326.28 Da
Type
Small molecule (not a peptide)
Amino Acid Sequence
C16H10F2N6O

Complex or non-standard sequence format

Metabolic Research

  • Obesity/Fat Loss

    Reduces body fat by increasing energy expenditure and fat oxidation without reducing food intake.

  • Insulin Resistance

    Research shows reversal of diet-induced insulin resistance in mouse models.

  • Metabolic Syndrome

    Addresses multiple components of metabolic syndrome through enhanced energy expenditure.

Combination Therapy Research

  • With Semaglutide/GLP-1 Agonists

    2024 research shows combining BAM-15 with semaglutide produces stronger metabolic benefits than either alone by countering metabolic adaptation.

  • Caloric Restriction Enhancement

    May help overcome weight loss plateaus by preventing metabolic adaptation/efficiency.

Other Research Areas

  • Liver Triglycerides

    Research shows decreased liver triglycerides in treated animals.

  • Glucose Tolerance

    Improved glucose tolerance observed in research models.

BAM-15 is orally bioavailable and is typically administered as an oral supplement in tablet or capsule form. Research in mice used dietary supplementation (0.1% of diet) or oral gavage dosing. Human dosing protocols are still being established through ongoing research. The compound has a relatively short half-life, which researchers are working to improve through structural modifications.

GoalDoseFrequencyRoute
Metabolic enhancement25-50 mgOnce or twice dailyOral
Research protocol (mouse equivalent)~10 mg/kgDailyOral gavage
Semaglutide/GLP-1 Agonists

2024 research shows combination produces stronger metabolic benefits by countering metabolic adaptation that limits GLP-1 efficacy.

synergistic
Tirzepatide

Similar synergy expected as with semaglutide; helps overcome weight loss plateaus.

synergistic
DNP/FCCP

Do not combine with other mitochondrial uncouplers - dangerous additive effects possible.

avoid
Thyroid hormones

Both increase metabolic rate; combination requires careful monitoring.

monitor
MOTS-c

Both affect mitochondrial function but through different mechanisms.

compatible
Hours

Increased oxygen consumption and metabolic rate

Days 1-7

Shift toward fat oxidation (lower respiratory exchange ratio)

Weeks 2-4

Measurable fat mass reduction in research

Weeks 4-8

Improved insulin sensitivity and glucose tolerance

Common Side Effects

  • Generally well-tolerated in research
  • Possible mild increase in body temperature (less than DNP)

Stop Signs - Discontinue if:

  • Significant hyperthermia/overheating
  • Excessive sweating
  • Rapid heart rate
  • Difficulty breathing

Contraindications

  • Hyperthyroidism or thyroid disorders
  • Heart conditions
  • Pregnancy or breastfeeding
  • Use of other mitochondrial uncouplers (DNP, FCCP)
  • Fever or active infection

Good Signs

  • White to off-white powder or tablets
  • Certificate of analysis with purity >98%
  • Proper packaging protected from light
  • Reputable supplier

Warning Signs

  • No third-party testing available
  • Unclear sourcing

Bad Signs

  • Discolored product
  • No purity information
  • Unknown origin
  • Mitochondrial uncoupler BAM15 reverses diet-induced obesity and insulin resistance in mice
    Nature Communications (2020)

    BAM15 is orally bioavailable, increases nutrient oxidation, decreases body fat mass without altering food intake, lean mass, body temperature, or markers of toxicity.

  • BAM15-mediated mitochondrial uncoupling protects against obesity and improves glycemic control
    EMBO Molecular Medicine (2020)

    BAM15 is 7-fold more potent than DNP, does not depolarize plasma membranes, and does not induce apoptosis at effective doses.

  • BAM15 as a mitochondrial uncoupler: a promising therapeutic agent for diverse diseases
    PMC Review (2023)

    Comprehensive review of BAM15 mechanisms including AMPK and PGC-1α activation pathways.

  • Beneficial effects of simultaneously targeting calorie intake and calorie efficiency in diet-induced obese mice
    Clinical Science (2024)

    Combining semaglutide and BAM15 produces stronger metabolic benefits than either alone; helps overcome metabolic adaptation.

Disclaimer

This information is for educational and research purposes only. Consult a healthcare professional before use.