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PE-22-28

Emerging

TREK-1 Channel Blocker | Shortened Spadin Analog

Dose 50-200mcg daily
Frequency Once daily
Cycle 4-8 weeks
Storage Refrigerate at 2-8°C, use within 4-6 weeks

Synthetic heptapeptide derived from Spadin positions 22-28, functioning as potent TREK-1 antagonist with enhanced selectivity and duration versus parent compound. Primary research focus on rapid antidepressant effects.

Mechanism of Action

Selectively blocks TREK-1 potassium channels (IC50: 0.12 nM). Enhances serotonin neurotransmission in dorsal raphe nucleus, triggering CREB activation and hippocampal neurogenesis.

Key Benefits

  • Rapid antidepressant effects within 4 days (preclinical)
  • Hippocampal neurogenesis and synaptogenesis
  • Enhanced serotonergic neurotransmission
  • Extended ~23 hour duration of action
  • 300-500x greater potency than full-length Spadin
Molecular Weight
773.89 Da
Chain Length
7 amino acids
Type
Linear heptapeptide
Amino Acid Sequence
One-letter: GVSWGL?
H₂N
H
G 1
O C
N
V 2
O C
N
S 3
O C
N
W 4
O C
N
H
G 5
O C
N
L 6
O C
N
H
? 7
COOH
Gly
1

Glycine

Position 1

Val
2

Valine

Position 2

Ser
3

Serine

Position 3

Trp
4

Tryptophan

Position 4

Gly
5

Glycine

Position 5

Leu
6

Leucine

Position 6

Arg
7

Arg (GVSWGLR)

Position 7

N-terminus C-terminus
Hydrophobic
Polar
Positive (+)
Negative (-)
Modified

Mental Health

  • Depression

    Primary research focus with rapid effects in behavioral models within 4 days.

  • Anxiety

    Anxiolytic properties demonstrated in preclinical anxiety models.

Neurogenesis

  • Hippocampal Neurogenesis

    Nearly doubles BrdU-positive cells after 4-day treatment.

  • Synaptogenesis

    Promotes new synapse formation through CREB activation.

Cognition

  • Memory Support

    Hippocampal and prefrontal cortex TREK-1 expression supports memory.

  • Neuroprotection

    Potential ischemic protection and neuronal survival support.

Subcutaneous injection to abdominal fat or thigh with site rotation.

GoalDoseFrequencyRoute
Antidepressant Effect50-200mcgOnce dailySubQ
Neurogenesis Support100-200mcgOnce dailySubQ

Reconstitution Instructions

Materials Needed:
  • PE-22-28 lyophilized powder
  • Bacteriostatic water
  • Insulin syringes
  • Alcohol swabs
  1. 1 Inject BAC water slowly down vial wall
  2. 2 Gently swirl (do not shake)
  3. 3 Store refrigerated 2-8°C
  4. 4 Use within 4-6 weeks
SSRIs (Fluoxetine, Sertraline)

Both enhance serotonergic pathways; monitor for excessive activity.

monitor
Semax/NA-Semax

Complementary neurogenesis and cognitive support.

synergistic
Selank

Different mechanisms for anxiety and mood support.

synergistic
Dihexa

Complementary neuroplasticity pathways.

synergistic
BPC-157

Different mechanisms, no contraindications.

compatible
MAOIs

Serotonin syndrome risk.

avoid
Days 1-4

Measurable antidepressant effects in preclinical models

Weeks 1-2

Neurogenesis and synaptogenesis processes established

Weeks 2-4

Potential mood and cognitive improvements emerge

Weeks 4-8

Sustained treatment allows full neurogenic effects

Common Side Effects

  • No effects on TREK-2, TRAAK, TASK-1 channels observed
  • No cardiac dysfunction or seizures in preclinical studies

Stop Signs - Discontinue if:

  • Serotonin syndrome signs
  • Severe persistent headaches
  • Cardiac symptoms
  • Seizure activity
  • Severe mood changes or suicidal ideation

Contraindications

  • Pregnancy and breastfeeding
  • Concurrent MAOI use

Good Signs

  • White to off-white lyophilized powder
  • Clear, colorless reconstituted solution
  • Certificate of Analysis with >98% purity
  • Proper cold-chain shipping

Warning Signs

  • Research compound only, not FDA-approved
  • Quality varies by supplier

Bad Signs

  • Cloudy, discolored, or particulate appearance
  • Clumped or sticky powder indicating moisture damage
  • Shortened Spadin Analogs Display Better TREK-1 Inhibition
    (2017)

    PE-22-28 IC50 0.12 nM vs 40-60 nM for Spadin; ~23 hour duration. Frontiers in Pharmacology.

  • Spadin: A Sortilin-Derived Peptide Targeting TREK-1 Channels
    (2010)

    Original discovery of TREK-1 blockade as antidepressant mechanism. PLOS Biology.

  • TREK-1 Deletion Results in Depression-Resistant Phenotype
    (2006)

    TREK-1 knockout mice show depression-resistant behavior in 5 tests. Nature Neuroscience.

Disclaimer

This information is for educational and research purposes only. Consult a healthcare professional before use.